IGF-1 LR3 vs MOTS-c

A detailed comparison to help you understand the differences and choose the right peptide for your research goals.

IGF-1 LR3

IGF-1 LR3 (Long R3 Insulin-like Growth Factor-1) is a modified version of IGF-1 with extended half-life and enhanced potency. The modifications prevent binding to IGF binding proteins, increasing bioavailability.

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MOTS-c

MOTS-c (Mitochondrial Open Reading Frame of the Twelve S rRNA type-c) is a mitochondrial-derived peptide that plays a key role in metabolic regulation and has emerged as a significant longevity research target.

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Side-by-Side Comparison

AspectIGF-1 LR3MOTS-c
MechanismBinds to IGF-1 receptors to promote protein synthesis, muscle growth, and fat metabolism. The LR3 modification (13 amino acid extension and arginine substitution) extends half-life from minutes to 20-30 hours.Activates AMPK pathway, enhances glucose uptake in skeletal muscle, improves insulin sensitivity, and regulates mitochondrial function. Acts as a metabolic hormone affecting whole-body energy homeostasis.
Typical DosageResearch protocols typically use 20-100mcg daily, often divided into multiple injections or administered bilaterally to target muscles.Research protocols typically use 5-10mg administered subcutaneously several times per week. Optimal dosing not yet established.
AdministrationIntramuscular injection (site-specific growth) or subcutaneous for systemic effects. Often cycled 4-6 weeks on, equal time off.Subcutaneous injection. Often combined with exercise protocols as it enhances exercise capacity and metabolic adaptation.
Side EffectsHypoglycemia, joint pain, water retention, potential jaw/hand growth with extended use, and injection site reactions.Limited human data. Animal studies show good tolerability. May affect energy levels and exercise performance.
Best For

What They Have in Common

Both IGF-1 LR3 and MOTS-c are commonly used for:

Key Differences

Unique to IGF-1 LR3:

Unique to MOTS-c:

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